complement cascade वाक्य
उदाहरण वाक्य
मोबाइल
- It participates in the complement cascade ending with cell death.
- Inhibition of the complement cascade can decrease this damage.
- Serum sickness is primarily caused by the antibodies clumping during infusion and triggering the complement cascade.
- These antibodies bind to antigens, making them easier targets for phagocytes, and trigger the complement cascade.
- LTA bound to targets can interact with circulating antibodies and activate the complement cascade to induce a passive immune kill phenomenon.
- The anaphylatoxins C3a, C4a, and C5a are cationic fragments generated during the complement cascade that participate in host defense.
- Eculizumab is a recombinant humanized monoclonal IgG2 / 4 antibody that selectively targets and inhibits the terminal portion of the complement cascade.
- Eculizumab specifically binds to the terminal Complement component 5, or C5, which acts at a late stage in the complement cascade.
- Activation of the complement cascade attracts phagocytes that leak peroxide and other reagents, which may increase the damage for the surviving heart cells.
- There are four different C5 convertases able to specifically convert alternative pathway ( "'C3bBbC3b "') of complement cascade.
- If the complement response is sufficient, red blood cells are damaged by the membrane attack complex, an effector of the complement cascade.
- Elements of the complement cascade can be found in many non-mammalian species including plants, birds, fish, and some species of invertebrates.
- The activation of the complement cascade can cause damage to cells, therefore the inhibition of the complement cascade can work as a medicine in certain conditions.
- The activation of the complement cascade can cause damage to cells, therefore the inhibition of the complement cascade can work as a medicine in certain conditions.
- Shiga-toxin directly activates the alternative complement pathway and also interferes with complement regulation by binding to complement factor H, an inhibitor of the complement cascade.
- Lending credibility to this has been the discovery of disease-associated genetic polymorphisms in other elements of the complement cascade including complement component 3 ( C3 ).
- In CDC, mAbs bind to specific antigen, leading to activation of the complement cascade, which in turn leads to formation of pores in tumor cells.
- It has been shown that CR1 can act as a negative regulator of the complement cascade, mediate immune adherence and phagocytosis and inhibit both the classic and alternative pathways.
- These points are of interest because C5a is generated by the interaction of bacteria with blood plasma components to activate the complement cascade which then cleave C5a from Complement component 5.
- All of the men were reported to have experienced cytokine release syndrome resulting in angioedema, swelling of skin and mucous membranes, akin to the effects of the complement cascade in severe allergic reaction.
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