| 21. | Gastrin acts on parietal cells directly and indirectly too, by stimulating the release of histamine.
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| 22. | Increased gastrin secretion also leads to peptic ulcers in > 50 % of MEN 1 patients.
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| 23. | The hydrogen potassium ATPase is activated indirectly by gastrin that causes ECL cells to release histamine.
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| 24. | Reduced gastrin secretion reduces acid secretion.
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| 25. | This results in an elevated gastrin level in an attempt to compensate for increased pH in the stomach.
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| 26. | Increased gastrin secretion increases gastric acid, which may inactivate pancreatic lipase, leading to diarrhea and steatorrhea.
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| 27. | The excitation of the vagus nerve, the administration of gastrin, secretin or cholecystokinin induce PP secretion.
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| 28. | Gastric secretion is stimulated chiefly by three chemicals : acetylcholine ( ACh ), histamine, and gastrin.
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| 29. | Gastrin stimulates acid secretion by directly stimulating parietal cells as well as by promoting histamine secretion by ECL cells.
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| 30. | The chief cells secrete pepsinogen in response to gastrin and especially Ach, and ACh also stimulates mucus secretion.
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