| 21. | Prominent suprasternal and carotid pulsations may be noted secondary to increased left ventricular stroke volume.
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| 22. | It is also possible to quantify the stroke volume in real-time on a beat-for-beat basis.
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| 23. | Therefore the stroke volume does not drop enough that it completely negates the increased heart rate.
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| 24. | In other words, if the end-diastolic volume increases, there is a corresponding increase in stroke volume.
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| 25. | This quantity is referred to as stroke volume.
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| 26. | This increased demand is met by an increase in the stroke volume of the octopus heart.
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| 27. | The decreased stroke volume causes a decreased forward cardiac output and an increase in the end-systolic volume.
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| 28. | Because stroke volume decreases in certain conditions and disease states, stroke volume itself correlates with cardiac function.
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| 29. | A reduced stroke volume may occur as a result of a failure of systole, diastole or both.
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| 30. | Because stroke volume decreases in certain conditions and disease states, stroke volume itself correlates with cardiac function.
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