Vasopressin has been found to inhibit the response of glomus cells to hypoxia, presumably because the usual response to hypoxia is vasodilatation, which in case of hypovolemia should be avoided.
22.
Other side effects may include headache, diarrhea, nausea, rash, jaw pain, vasodilatation, dizziness, edema ( swelling ), pruritus ( itching ), and hypotension.
23.
This happens due to the widespread peritoneal irritation by the ruptured viscus contents, as in peptic ulcer perforation, with consequent strong vagal activation, and generalized, extensive peripheral vasodilatation and bradicardia.
24.
The first one is caused by the vasodilatation of the cerebral blood vessels produced by the hypoxia; the second one is caused by the vasoconstriction of the pulmonary arterioles, caused by the hypoxia.
25.
Blood vessels are under sympathetic tone, therefore the release of noradrenaline and adrenaline will cause vasoconstriction of non-essential tissues such as the liver, intestines, and kidneys, and decrease neurotransmitter release to the active muscles promoting vasodilatation.
26.
The mechanism for ?-adrenoceptor blocker-associated adverse events is generally ascribed to inhibition of ?2-adrenoceptor-mediated vasodilatation, leaving ?1-adrenoceptor-mediated vasoconstrictor responses to catecholamines unopposed and, thus, severe and potentially refractory hypertension.
27.
He performed extensive research involving blood pressure, blood viscosity, intracranial circulation, blood supply of organs, vasodilatation, and a phenomenon he called " Windkesseleffekt ", of which he demonstrated plays an important role in the maintenance of blood pressure.
28.
Under certain conditions, this can backfire : Fulminant infection ( sepsis ) causes excess production of nitric oxide by macrophages, leading to vasodilatation ( widening of blood vessels ), probably one of the main causes of hypotension ( low blood pressure ) in sepsis.
29.
Within the autoregulatory range, as CPP falls there is, within seconds, vasodilatation of the cerebral resistance vessels, a fall in cerebrovascular resistance and a rise in cerebral-blood volume ( CBV ), and therefore CBF will return to baseline value within seconds ( see as ref.
30.
The vasodilatation caused by sodium nitroprusside has been shown to be mediated not only by NO generation, but also by endogenous cyanide generation, which adds not only toxicity, but also some additional antihypertensive efficacy compared to nitroglycerine and other non-cyanogenic nitrates which do not cause blood cyanide levels to rise.
