| 31. | The presence of gastrin stimulates parietal cells of the stomach to secrete hydrochloric acid ( HCl ) / gastric acid.
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| 32. | Additionally, elevated gastrin levels may be present in chronic gastritis resulting from " H pylori " infection.
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| 33. | Gastrin is transferred from a specific type of G cell in the gastric epithelium to the ECL cells by blood.
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| 34. | Vagal stimulation of the peptidergic neurons, occurring simultaneously, leads to the release of gastrin-releasing-peptide.
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| 35. | "' Little gastrin I "'is a form of gastrin commonly called as gastrin-17.
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| 36. | "' Little gastrin I "'is a form of gastrin commonly called as gastrin-17.
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| 37. | "' Little gastrin I "'is a form of gastrin commonly called as gastrin-17.
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| 38. | Eventually, all the parietal cells are lost and achlorhydria results leading to a loss of negative feedback on gastrin secretion.
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| 39. | Other possible mechanisms include increased vagal activity and secretion of insulin and gastrin as well as improved parent-infant relationships.
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| 40. | D cells in the stomach contain CCKBR ( which respond to gastrin ) and M3 receptors ( which respond to Ach ).
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