| 31. | The distinguishing feature of intravascular hemolysis is the release of RBC contents into the blood stream.
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| 32. | Increased amounts of bilirubin are formed in hemolysis, which generates increased urobilinogen in the gut.
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| 33. | No evidence for hemolysis is seen.
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| 34. | A glutathione deficiency, congenital or acquired, can lead to hemolysis caused by oxidative stress.
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| 35. | Lactate dehydrogenase is a marker of hemolysis and is elevated ( > 600 U / l ).
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| 36. | Despite causing some severe pathologies, lots of cases of hemolysis do not suppose a health hazard.
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| 37. | It has also been shown that PAMAM dendrimers cause rupturing of red blood cells, or hemolysis.
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| 38. | In intravascular hemolysis, free hemoglobin will be released into circulation and hence haptoglobin will bind the hemoglobin.
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| 39. | In hereditary spherocytosis, red blood cells fail to pass through and get phagocytosed, causing extravascular hemolysis.
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| 40. | It is more often the result of intravascular hemolysis, which occurs in about half of all cases.
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