| 31. | When the action potential reaches the sarcoplasmic reticulum it triggers the release of Ca2 + from the Ca2 + channels.
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| 32. | The sarcoplasmic reticulum surrounds the myofibrils and holds a reserve of the calcium ions needed to cause a muscle contraction.
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| 33. | Concentrations of intracellular calcium can be increased by increasing influx into the cell or stimulating release from the sarcoplasmic reticulum.
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| 34. | At high heart rates, phospholamban is phosphorylated and deactivated thus removing most from the cell back into the sarcoplasmic reticulum.
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| 35. | The sarcoplasmic reticulum, a specialized type of smooth endoplasmic reticulum, forms a network around each myofibril of the muscle fiber.
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| 36. | T tubules are the pathways for action potentials to signal the sarcoplasmic reticulum to release calcium, causing a muscle contraction.
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| 37. | This depolarisation triggers the entry of calcium from the sarcoplasmic reticulum to cause contraction ( tensing ) of the muscle.
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| 38. | It is found that there is an increase with calcium reuptake because of changes within sarcoplasmic reticulum morphology and structure.
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| 39. | The so-called delayed after-depolarizations are thought to be due to an increased Ca 2 + filling of the sarcoplasmic reticulum.
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| 40. | In March 1902, he provided the first accurate description of the reticular network ( sarcoplasmic reticulum ) in skeletal muscle fibers.
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