After their release from the sarcoplasmic reticulum, calcium ions interact with contractile proteins that utilize ATP to shorten the muscle fiber.
42.
The inward flow of calcium from the L-type calcium channels activate ryanodine receptors to release calcium ions from the sarcoplasmic reticulum.
43.
Ryanodine receptors mediate the release of calcium ions from the sarcoplasmic reticulum and endoplasmic reticulum, an essential step in muscle contraction.
44.
The concentration of calcium within muscle cells is controlled by the sarcoplasmic reticulum, a unique form of endoplasmic reticulum in the sarcoplasm.
45.
Additionally, the release of calcium ions from the sarcoplasmic reticulum during muscle contraction inactivates the inhibitory ? subunit and activates PhK fully.
46.
One of the components is a membrane called the sarcoplasmic reticulum, which holds the calcium-uptake sites and little pouches of calcium ions.
47.
Nitroxyl interacts with RyR2 to increase the probability of Ryanodine receptor opening, thereby enhancing Ca 2 + release from the sarcoplasmic reticulum.
48.
Very quickly Ca2 + is actively transported back into the sarcoplasmic reticulum, which blocks the interaction between the thin and thick filament.
49.
They cause an increased calcium release from the sarcoplasmic reticulum of muscle cells which can result in rigidity and eventual cell breakdown.
50.
The release of Ca 2 + from the sarcoplasmic reticulum causes an increase in the concentration of Ca 2 + in the cytosol.
