| 1. | Hemosiderin pigment deposition due to vessel rupture may be observed.
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| 2. | Long-standing lesions may show hemosiderin-laden macrophages and calcifications.
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| 3. | Hemosiderin can accumulate in different organs in various diseases.
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| 4. | Hemosiderin may deposit in diseases associated with iron overload.
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| 5. | If the systemic iron overload is corrected, over time the hemosiderin is slowly resorbed by macrophages.
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| 6. | The tissue remnant usually includes fibrous tissue and signs of old infarction with hemosiderin deposition identified histologically.
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| 7. | Alveolar macrophages ( dust cells ) engulf the red blood cells, and become engorged with brownish hemosiderin.
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| 8. | The subretinal exudate consists of cholesterol crystals, macrophages laden with cholesterol and pigment, erythrocytes, and hemosiderin.
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| 9. | Other zonal injury patterns include zone I deposition of hemosiderin in hemochromatosis and zone II necrosis in yellow fever.
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| 10. | This results in red cells breaking down, with iron containing hemosiderin possibly contributing to the pathology of this entity.
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