endosome वाक्य
उदाहरण वाक्य
मोबाइल
- D12, the mouse homolog of MDS032, is a Golgi secretory apparatus and with endosome-lysosome transport.
- Rab5 contributes to early endosome fusion that works through the coordination of other effector proteins, as mentioned above.
- It is because of these vesicles that the endosome is sometimes known as a " multivesicular body ".
- The major function of the gp64 envelope protein is to cause the pH-mediated envelope fusion to the endosome.
- The pH in the late endosome can reach 6.0, which causes dissociation of M6P from its receptor.
- Inside the endosome, the toxin is split by a trypsin-like protease into its individual A and B fragments.
- This gene encodes a protein involved in endosomal sorting of cell surface receptors via a multivesicular body / late endosome pathway.
- Exosomes are formed by invagination within a cell to create an intracellular vesicle called an endosome, or an endocytic vesicle.
- The cell then attempts to begin digesting the contents of the endosome by acidifying its interior and transforming it into a lysosome.
- The MPRs are packed into vesicles that bud off the late endosome and return to the " trans "-Golgi network.
- See ubiquitination, proteasome, autophagy, endosome, lysosome . . . no doubt I'm forgetting lots of biggies.
- The second two involve the full fusion of the vesicle with the membrane, followed by recycling, or recycling into the endosome.
- Below a pH of 6.0 ( the pH in an endosome ), both LF and EF become disordered molten globules.
- The dropping pH inside the endosome frees the outer domain of E1 and causes the fusion of the viral envelope with the endosomal membrane.
- The sequential stages of HSV entry are analogous to endocytosed after binding to the receptors, and the fusion could occur at the endosome.
- The endosome is moved to the Golgi apparatus, where the A1 protein is recognized by the endoplasmic reticulum chaperon, protein disulfide isomerase.
- TRPML1 is believed to channel iron ions across the endosome / lysosome membrane into the cell and so its malfunction causes cellular iron deficiency.
- The cellular membrane pinches in a procession known as pinocytosis and allows entry of the virus into the cell by way of an endosome.
- The believed mechanism for this has to do with an acidification of the endosome, similar to what is done in the influenza virus.
- The low pH within the endosome then triggers BTV's membrane penetration protein VP5 to undergo a conformational change that disrupts the endosomal membrane.
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