hyperalgesia वाक्य
उदाहरण वाक्य
मोबाइल
- Spinal Aloxe3, apparently through its ability to make hepoxilins, appears responsible for the hyperalgesia which accompanies inflammation in rats.
- Some of the released mediators such as bradykinin increase the sensitivity to pain ( hyperalgesia, " dolor " ).
- Previously hyperalgesia was implied, since hyperesthesia was mentioned in the previous note and hyperalgesia is a special case of hyperesthesia ".
- Previously hyperalgesia was implied, since hyperesthesia was mentioned in the previous note and hyperalgesia is a special case of hyperesthesia ".
- The definition also complies with a note which is : " It may occur with allodynia, hyperesthesia, hyperalgesia, or dysesthesia.
- It is often suggested hyperalgesia and allodynia assist organisms to protect themselves during healing, but experimental evidence to support this has been lacking.
- Various studies of humans and animals have demonstrated that primary or secondary hyperalgesia can develop in response to both chronic and acute exposure to opioids.
- Although tolerance and opioid-induced hyperalgesia both result in a similar need for dose escalation, they are nevertheless caused by two distinct mechanisms.
- This manifests as an increased sensitivity to painful stimuli ( hyperalgesia ) or pain sensation in response to non-painful stimuli ( allodynia ).
- One major pathway being through stimulation of the nociceptin receptor, and blocking this receptor may therefore be a means of preventing the development of hyperalgesia.
- It does not have analgesic effects in its own right, but prevents the development of hyperalgesia, and also shows antidepressant effects in animal studies.
- It has been used to research the involvement of the 5-HT 7 receptor in as yet poorly understood processes such as allodynia and hyperalgesia.
- Identifying the development of hyperalgesia is of great clinical importance since patients receiving opioids to relieve pain may paradoxically experience more pain as a result of treatment.
- Ablation of ?-opioid-expressing neurons in the RVM with a dermorphin-saporin conjugate eliminated the allodynia and hyperalgesia caused by a nerve injury.
- Recent evidence for generalized increased pain sensitivity or hyperalgesia in CTTH strongly suggests that pain processing in the central nervous system is abnormal in this primary headache disorder.
- He found that among the cytokines, interleukin 1b mediates the endogenous release of prostaglandins and IL-8 is responsible for the development of the sympathetic hyperalgesia.
- This abnormal central sensitization cycle results in increased pain ( hyperalgesia ) and pain responses from previously non-noxious stimuli evoke a pain response ( allodynia ).
- Injection of the CCK-saporin conjugate also reversed allodynia and hyperalgesia in a nerve injury model, producing the same results as the dermorphin-saporin conjugate.
- The suggestion that chronic pain patients who are diagnosed as experiencing opioid-induced hyperalgesia ought to be completely withdrawn from opioid therapy has also been met with criticism.
- Various tolerance to opioid analgesic drugs, and in the emergence of side-effects such as hyperalgesia and allodynia that can become a problem following extended use of opioid drugs.
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