steroidogenesis वाक्य
उदाहरण वाक्य
मोबाइल
- The first step of steroidogenesis is dependent on the transfer of electrons from NADPH to a P450 type enzyme ( P450scc ) via an electron-transfer chain that includes two additional proteins.
- Thus, MLN64 is also proposed to move cholesterol inside the mitochondria under certain conditions to initiate StAR-independent steroidogenesis, such as in the human placenta which lacks StAR yet produces steroids.
- Depending upon the tissue, it was shown to be involved in steroidogenesis, haem biosynthesis, apoptosis, cell growth and differentiation, complex with the voltage-dependent anion channel ( VDAC)
- In this field, Israel organized the first International Symposium in Molecular Steroidogenesis in Jerusalem in 1991 which served as the cornerstone for a continuing series of international symposia for scientists who specialize in this field.
- Apparently to cope with the radicals produced by this system and by enhanced mitochondrial metabolism, the levels of antioxidant enzymes catalase and superoxide dismutase also increase in parallel with the enhanced steroidogenesis in the corpus luteum.
- These are adrenal dysgenesis ( the gland has not formed adequately during development ), impaired steroidogenesis ( the gland is present but is biochemically unable to produce cortisol ) or adrenal destruction ( disease processes leading to glandular damage ).
- Also in contrast to, bicalutamide neither inhibits nor suppresses androgen production in the body ( i . e ., it does not act as an antigonadotropin or steroidogenesis inhibitor ), and instead exclusively mediates its antiandrogen effects by blocking androgen receptor activation at the level of the.
- Gas chromatography / mass spectrometry analysis of urinary steroids from pregnant women carrying a POR-deficient fetus described in an earlier report also supports the existence of this pathway, and the relevance of the " backdoor " pathway along with POR dependent steroidogenesis have become clearer from recent studies.
- "' Congenital adrenal hyperplasia "'( "'CAH "') are any of several autosomal recessive diseases resulting from mutations of genes for enzymes mediating the biochemical steps of production of mineralocorticoids, glucocorticoids or sex steroids from cholesterol by the adrenal glands ( steroidogenesis ).
- "' FGIN-1-27 "'is an anxiolytic drug which acts as a selective agonist at the peripheral benzodiazepine receptor, also known as the mitochondrial 18 kDa translocator protein or TSPO . It is thought to produce anxiolytic effects by stimulating steroidogenesis of neuroactive steroids such as allopregnanolone.
- Like other NSAAs such as flutamide and bicalutamide, nilutamide, without concomitant GnRH analogue therapy, increases serum androgen ( by two-fold in the case of testosterone ), estrogen, and prolactin levels due to inhibition of AR-mediated suppression of steroidogenesis via negative feedback on the hypothalamic-pituitary-gonadal axis.
- "' SSR-180, 575 "'is a drug which acts as a selective agonist at the peripheral benzodiazepine receptor, also known as the mitochondrial 18 kDa translocator protein or TSPO . It has been shown to have neuroprotective and cardioprotective effects and to stimulate steroidogenesis of pregnenolone in the brain, which may be linked to its neuroprotective action.
- "' FGIN-1-43 "'is an anxiolytic drug which acts as a selective agonist at the peripheral benzodiazepine receptor, also known as the mitochondrial 18 kDa translocator protein or TSPO . It is thought to produce anxiolytic effects by stimulating steroidogenesis of neuroactive steroids such as allopregnanolone, and is several times more potent than the related drug FGIN-127.
- The results suggest that trophic hormones ( e . g ., leutenizing hormone, adrenocorticotropic hormone ) stimulate these steroid producing cells to make 5-( " S " )-HETE and 5-( " S " ) HpEPE which in turn increase the synthesis of steroidogenic acute regulatory protein; the latter protein promotes the rate-limiting step in steroidogenesis, transfer of cholesterol from the outer to the inner membrane of mitochondria and thereby acts in conjunction with trophic hormone-induce activation of protein kinase A to make progesterone and testosterone.
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