hyperexcitability वाक्य
उदाहरण वाक्य
मोबाइल
- Hyperexcitability, a characteristic feature of epileptogenesis in which the likelihood that neural networks will be activated is increased, may be due to loss of inhibitory neurons that would normally balance out the excitability of other neurons, such as GABA A receptors to the neurotransmitter may decrease, resulting in less inhibition.
- Loss of KCC2 following neuronal damage ( i . e . ischemia, spinal cord damage, physical trauma to the central nervous system ) results in the loss of inhibitory regulation and the subsequent development of neuronal hyperexcitability, motor spasticity, and seizure-like activity as GABA A receptors and glycine receptors revert from hyperpolarizing to depolarizing postsynaptic effects.
- This competition may partially attenuate NMDA receptor and channel mediated hyperexcitability after seizures . It is unlikely that these effects would be long-lasting, but by decreasing the effectiveness of NMDA-type glutamate receptors, high H + concentrations could increase the threshold needed to excite the cell, inhibiting the seizure and potentially slowing neuronal signaling after the event.
- In general, high titers ( usually > 400 pM ) are more frequently associated with defined clinical phenotypes ( mainly LE, but also peripheral nerve hyperexcitability ( PNH ), seizures, or MoS ), whereas low titers of Abs can be found in patients with either or both CNS and PNS syndromes but are also detected in patients without a clear immune-mediated phenotype.
- Certain studies suggested a link between sporadic ALS, specifically in athletes, and a diet enriched with branched-chain amino acids, a common dietary supplement among athletes, which cause cell hyperexcitability resembling that usually observed in people with ALS . The proposed underlying mechanism is that cell hyperexcitability results in increased calcium absorption by the cell, and thus brings about cell death of neuronal cells, which have particularly low calcium buffering capabilities.
- Certain studies suggested a link between sporadic ALS, specifically in athletes, and a diet enriched with branched-chain amino acids, a common dietary supplement among athletes, which cause cell hyperexcitability resembling that usually observed in people with ALS . The proposed underlying mechanism is that cell hyperexcitability results in increased calcium absorption by the cell, and thus brings about cell death of neuronal cells, which have particularly low calcium buffering capabilities.
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