long chain fatty acid वाक्य
उदाहरण वाक्य
मोबाइल
- This deficiency falls within a class of disorders called peroxisome biogenesis disorders ( PBDs ), although it is quite different from other peroxisomal disorders and does not share classic DHCA and EHCA and to a lesser extent Very long chain fatty acid and phytanic acid.
- This is due to the fact that the beta oxidation of long chain fatty acids with an odd number of carbons produces propionyl-CoA . Most part of this metabolite is transformed into succinyl-CoA, which is the precursor of the tricarboxylic acid cycle.
- LA is the most abundant fatty acid in the skin epidermis, being present mainly esterified to the omega-hydroxyl residue of amide-linked omega-hydroxylated very long chain fatty acids ( VLCFAs ) in a unique class of ceramides termed esterified omega-hydroxyacyl-sphingosine ( EOS ).
- Oxidative rancidity occurs when long chain fatty acids ( like those found in soybean oil ) become oxidized due to certain conditions such as light, heat, oxygen, heavy metals, etc . It involves three steps, initiation, propagation and termination, and it results in the formation of off-flavors, off-aromas, and compounds potentially hazardous to human health.
- Each type of leukodystrophy has a unique pathophysiology, but all five of these in some way affect a subset of glial cells, therefore disrupting myelin production and maintenance, and usually involve a mutation involving genes that code for enzymes necessary for the catabolism of very long chain fatty acids ( VLCFAs ) that are toxic to the myelin-producing cells of the central nervous system.
- CYP2U1 hydroxylates arachidonic acid, docosahexaenoic acid ( DHA ), and other long chain fatty acids at their terminal ( i . e ., ? ) carbon to from 20-hydroxy-arachidonic acid ( i . e . 20-Hydroxyeicosatetraenoic acid or 20-HETE ), 22-hydroxy-docosahexaneoic acid, and other ?-hydroxy long chain fatty acids, respectively, plus lesser amounts of these fatty acids ?-1 hydroxy metabolites, i . e . 19-HETE, 21-hydroxy-docosahexaenoic acid, and other ?-1 hydroxy long chain fatty acids, respectively.
- CYP2U1 hydroxylates arachidonic acid, docosahexaenoic acid ( DHA ), and other long chain fatty acids at their terminal ( i . e ., ? ) carbon to from 20-hydroxy-arachidonic acid ( i . e . 20-Hydroxyeicosatetraenoic acid or 20-HETE ), 22-hydroxy-docosahexaneoic acid, and other ?-hydroxy long chain fatty acids, respectively, plus lesser amounts of these fatty acids ?-1 hydroxy metabolites, i . e . 19-HETE, 21-hydroxy-docosahexaenoic acid, and other ?-1 hydroxy long chain fatty acids, respectively.
- CYP2U1 hydroxylates arachidonic acid, docosahexaenoic acid ( DHA ), and other long chain fatty acids at their terminal ( i . e ., ? ) carbon to from 20-hydroxy-arachidonic acid ( i . e . 20-Hydroxyeicosatetraenoic acid or 20-HETE ), 22-hydroxy-docosahexaneoic acid, and other ?-hydroxy long chain fatty acids, respectively, plus lesser amounts of these fatty acids ?-1 hydroxy metabolites, i . e . 19-HETE, 21-hydroxy-docosahexaenoic acid, and other ?-1 hydroxy long chain fatty acids, respectively.
- Within the active site the negatively charged oxygen on the fatty acid attacks the alpha phosphate on ATP, forming an ATP-long chain fatty acid intermediate . ( Step 1, Figure 3 ) In the second step, Pyrophosphate ( PPi ) leaves, resulting in an AMP-long chain fatty acid molecule within the enzyme s active site . ( Step 2, Figure 3 ) Coenzyme A now enters the enzyme and another intermediate is formed which consists of AMP-long chain fatty acid-Coenzyme A . ( Step 3, Figure 3 ) At the end of this mechanism two products are released, AMP and acyl coa synthetase . ( Step 4, Figure 3)
- Within the active site the negatively charged oxygen on the fatty acid attacks the alpha phosphate on ATP, forming an ATP-long chain fatty acid intermediate . ( Step 1, Figure 3 ) In the second step, Pyrophosphate ( PPi ) leaves, resulting in an AMP-long chain fatty acid molecule within the enzyme s active site . ( Step 2, Figure 3 ) Coenzyme A now enters the enzyme and another intermediate is formed which consists of AMP-long chain fatty acid-Coenzyme A . ( Step 3, Figure 3 ) At the end of this mechanism two products are released, AMP and acyl coa synthetase . ( Step 4, Figure 3)
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