| 1. | These changes impair mitochondrial energy production and drive cardiac myocyte apoptosis.
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| 2. | GATA4 and GATA6 also play a role in myocyte differentiation.
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| 3. | TEAD proteins and MEF2 ( myocyte enhancer factor 2 ) interact physically.
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| 4. | These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation.
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| 5. | This means that the refractoriness of the ventricular myocyte increases at lower heart rates.
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| 6. | The electrical depolarizations that trigger cardiac myocytes to contract arise spontaneously within the myocyte itself.
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| 7. | One TC establishing contacts with a myocyte by a Tp of about 65 mm long.
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| 8. | The cardiac conduction system ( and AV node part of it ) coordinates myocyte mechanical activity.
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| 9. | Excitation of a myocyte causes depolarization at its synapses, the neuromuscular junctions, which triggers action potential.
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| 10. | ITM2A is also involved in activation of T-cells in the immune system and in myocyte differentiation.
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