| 1. | Aspirin inhibits platelet aggregation by inhibiting the action of thromboxane A 2.
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| 2. | This isomerization mechanism shows prostaglandin H2 being converted to thromboxane.
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| 3. | Thromboxane A2 ( TX2 ) has a positive feedback in platelet activation.
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| 4. | Receptors that mediate TXA2 actions are thromboxane A2 receptors.
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| 5. | The prototype for these drugs is aspirin, which inhibits the production of thromboxane.
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| 6. | Picotamide has activity both as a thromboxane synthase inhibitor and as a thromboxane receptor antagonist.
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| 7. | Picotamide has activity both as a thromboxane synthase inhibitor and as a thromboxane receptor antagonist.
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| 8. | Thromboxane A ( TXA ) is derived from the prostaglandin H2 ( PGH2 ) molecule.
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| 9. | An olive oil fraction containing DHPE can inhibit platelet aggregation and thromboxane B2 formation in vitro.
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| 10. | Thromboxane A2 is also a known vasoconstrictor and is especially important during tissue injury and inflammation.
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